Lectures

Diabetes Insipidus (DI)

by Carlo Raj, MD
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    About the Lecture

    The lecture Diabetes Insipidus (DI) by Carlo Raj, MD is from the course (Nephrogenic) Diabetes Insipidus (DI). It contains the following chapters:

    • Nephrogenic Diabetes Insipidus
    • Etiology of Nephrogenic Diabetes Insipidus
    • Actions of ADH
    • Management of Nephrogenic Diabetes Insipidus
    • Differentials

    Included Quiz Questions

    1. Diabetes mellitus type I
    2. Diabetes mellitus type II
    3. Myasthenia gravis
    4. Androgen Insensitivity syndrome
    5. Pseudohypoparathyroidism
    1. Aquaporin2 channels on principal cell membrane
    2. Urine volume
    3. Clearance of water
    4. Plasma ADH concentration
    5. Plasma osmolarity
    1. Release of ADH is inhibited from the posterior pituitary.
    2. Urine osmolarity is decreased.
    3. Clearance of free water will be positive.
    4. It may be induced by administration of demeclocycline.
    5. Water reabsorption is decreased in the distal tubules.
    1. Receptor and/or subsequent signaling pathway defect.
    2. Insufficient production of hormone.
    3. Excessive negative feedback.
    4. Insufficient precursors for hormone production.
    5. None are correct.
    1. Aquaporin2 gene mutation
    2. Arginine vasopressin 2 receptor gene mutation
    3. Factor VIII gene mutation
    4. Von Willebrand factor gene mutation
    5. V1 receptor mutation
    1. Renal tubular necrosis
    2. Demeclocycline administration
    3. Arginine vasopressin 2 receptor gene mutation
    4. Aquaporin 2 gene mutation
    5. Chronic lithium use
    1. Epithelial sodium channel on the principal cell
    2. NaCl symport on the principal cell
    3. ROMK channel on the intercalated cell
    4. Na-H+ antiport on the intercalated cell
    5. Na-K+ ATPase on the principal cell
    1. Hemophilia A
    2. Hemophilia B
    3. Thrombocytopenia
    4. Von Willebrand disease type III
    5. Bernard-Soulier syndrome
    1. Cyclic AMP
    2. ATP
    3. Adenylate cyclase
    4. Calcium
    5. Phosphodiesterase
    1. Vasodilation of blood vessels.
    2. Increased permeability to urea in the distal tubules.
    3. Increased reabsorption of water in the distal tubules.
    4. Release of Factor VIII from endothelium.
    5. Release of von willebrand factor from endothelium.
    1. Phosphodiesterase
    2. Protein Kinase A
    3. None of these are correct.
    4. Na-K-ATPase
    5. Adenylate cyclase
    1. Prostaglandin E2 receptor agonist
    2. Demeclocycline
    3. Acetaminophen
    4. COX2 inhibitors
    5. Cryoprecipitate
    1. Functioning Factor VIII levels
    2. Plasma osmolarity
    3. Platelet count
    4. Bleeding time
    5. Partial thromboplastin time
    1. Dilutional hyponatremia
    2. Osmotic diuresis
    3. Central Pontine myelinolysis
    4. Hypovolemia
    5. Cell swelling and lysis
    1. Amiloride - ENac
    2. Furosemide – NaK2Cl cotransporter
    3. Thiazides – ENac
    4. Amiloride – NaCl symporter
    5. Thiazides – NaCl symporter
    1. Prevent osmotic diuresis
    2. Improve nutritional status
    3. Increase the urine output
    4. Induce weight loss
    5. Prevent hypertension
    1. Furosemide
    2. Hydrochlorothiazide
    3. Spirinolactone
    4. Amiloride
    5. ACE inhibitors
    1. Central diabetes insipidus
    2. Hypocalcemia
    3. Nephrogenic diabetes insipidus
    4. Hypertension in the elderly
    5. Hypertension in African Americans
    1. SIADH
    2. Central diabetes insipidus
    3. Water drinking
    4. Water deprivation
    5. Nephrogenic diabetes insipidus
    1. Free water clearance
    2. Urine osmolarity
    3. Thirst
    4. Serum ADH concentration
    5. Plasma osmolarity
    1. Blood pressure
    2. Plasma renin activity
    3. Urine Ca concentration
    4. Serum magnesium concentration
    5. Plasma aldosterone concentration
    1. Bartter syndrome
    2. Gitelman syndrome
    3. Conn syndrome
    4. SIADH
    5. Liddle syndrome
    1. Liddle syndrome
    2. SIADH
    3. Bartter syndrome
    4. Reninoma
    5. Conn syndrome

    Author of lecture Diabetes Insipidus (DI)

     Carlo Raj, MD

    Carlo Raj, MD


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