Nephritic Syndrome and Forms of Glomerulonephritis

by Carlo Raj, MD

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    About the Lecture

    The lecture Nephritic Syndrome and Forms of Glomerulonephritis by Carlo Raj, MD is from the course Glomerulonephritis. It contains the following chapters:

    • Nephritic Disorders: Glomerulonephritis
    • Post-streptococcal Glomerulonephritis
    • Non-streptococcal Acute Glomerulonephritis
    • Diffuse Proliferative Glomerulonephritis
    • Subendothelial IC Deposition Viewed With EM

    Included Quiz Questions

    1. Deposits are subepithelial.
    2. Deposits are between the endothelium and the GBM.
    3. Deposits activate T cell cytokines causing glomerular damage.
    4. Deposits may also be found in the heart tissue leading to acute rheumatic fever.
    5. Immune complexes activate the classical complement pathway.
    1. Hypoalbuminemia
    2. Hematuria
    3. RBC casts in the urine
    4. Hypernatremia
    5. Periorbital edema
    1. Anti-DNAse B antibodies
    2. Anti-streptolysin
    3. Anti-Nicotinamide-adenine-dinucleotidase
    4. Anti-streptokinase
    5. Anti-hyaluronidase
    1. Serum C3
    2. RBC casts
    3. Serum albumin
    4. Azotemia
    5. Proteinuria
    1. Rapidly progressive glomerulonephritis
    2. Acute tubular necrosis
    3. Acute interstitial nephritis
    4. Rheumatic fever
    5. Acute renal failure
    1. Hematuria presents 1-3 days following a strep infection.
    2. Treatment of streptococcal infection is often curative.
    3. The strain responsible for causing glomerulonephritis will never cause acute rheumatic fever.
    4. Hypertension is often transient.
    5. More than 95% of children recover completely
    1. Subendothelial
    2. Subepithelial
    3. More than one answer is correct
    4. Intermembranous
    5. Mesangial
    1. Both involve activation of the alternative complement pathway.
    2. Both involve immune complex deposition.
    3. Both show granular pattern on immunofluorescence.
    4. Both may present with between 150 mg-3.5 g/day of protein in the urine.
    5. Both present with hematuria.
    1. Anti-dsDNA antibodies
    2. Anti-streptokinase
    3. Anti-Ro antibodies
    4. Anti-smith antibodies
    5. Anti-DNase B antibodies
    1. Corticosteroids and cyclophosphamide
    2. Corticosteroids and NSAIDs
    3. Watchful waiting, it usually resolves on its own.
    4. ACE inhibitors and cyclophosphamide
    5. Dialysis is the only treatment
    1. It is the only type of glomerulonephritis caused by SLE.
    2. It is Associated with “wire looping” of capillaries.
    3. It often evolves into chronic renal failure.
    4. Hypercellularity of the glomerulus is seen on light microscopy.
    5. Serum ANA test has a “rim” pattern.

    Author of lecture Nephritic Syndrome and Forms of Glomerulonephritis

     Carlo Raj, MD

    Carlo Raj, MD

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