by Carlo Raj, MD

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    If that was hyperaldosteronism, we will then move on to hypoaldosteronism. You do the same thing, quickly tell me. Serum levels decreased, potassium levels elevated, hydrogen is increased, thus Ph is decreased. Can you do it that quickly? Yes, you can. Listen to me again, you will get it. Results in… results from type 4 renal tubular acidosis or could result in. What do you mean? I just told you, if you didn’t have enough aldosterone, your hydrogen concentration increases in your plasma, what happens to your Ph? Oh, renal tubular acidosis. When you say renal tubular acidosis, what is that acidosis actually referring to? Not in the urine, right, it is in the plasma. Type 4, type 4, type 4. Remember, RTA, you want to know 1, 2, and 4. However, the biggest difference between this type of RTA and any other, if you don’t have aldosterone, take your time, aldosterone and potassium. If you don’t have aldosterone, you are not getting rid of potassium, you are retaining it resulting in hyperkalemia. Hmm, that is dangerous, that hyperkalemia is dangerous, especially for the heart. You are then causing, as you remember, depolarization of your resting membrane potential and that is not a good thing, is it? Hypoaldosteronism hyporeninemic, how is this occurring? Common in chronic kidney disease, so if the kidney starts decreasing in function then you know that you don’t have enough renin. If you don’t have enough renin then what then happens to your aldosterone level? Decreases. This is called hyporeninemic hypoaldosteronism. Say that three times fast. Language, language, language, apart from may be a little bit of memorization, but really understand what this is saying. Also seen with NSAIDs, cyclosporin and HIV, you could potentially have hyporeninemic hypoaldosteronism. Hyperreninemic hypoaldosteronism, how is this even possible? What...

    About the Lecture

    The lecture Hypoaldosteronism by Carlo Raj, MD is from the course Adrenal Gland Disorders.

    Included Quiz Questions

    1. Hypercalcemia
    2. Hyperkalemia
    3. Metabolic acidosis
    4. Suppression of ammonia excretion
    5. Hyponatremia
    1. ACE inhibitor therapy
    2. Chronic kidney disease
    3. Chronic diabetes
    4. Cyclosporin and NSAID use
    5. HIV
    1. TTPK
    2. FF/GFR
    3. Urine osmolarity/serum osmolarity
    4. Cockcroft-Gault equation
    5. Urine K/Serum K
    1. Hyporeninemic hypoaldosteronism
    2. Hypereninemia hypoaldosteronism
    3. Congenital isolated hypoaldosteronism
    4. Congenital adrenal hyperplasia
    5. Primary adrenal insufficiency

    Author of lecture Hypoaldosteronism

     Carlo Raj, MD

    Carlo Raj, MD

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