Lectures

Tuberculous Meningitis

by Carlo Raj, MD
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    We’re moving on to tuberculous meningitis. So tuberculosis. And so when you say tuberculosis meningitis, keep in mind that granted, it is bacterial. However -- However, I need you to keep this separate from the other bacterial causes that I’d walk you through based on the age groups Are you with me? Remember that table that I gave you where it was predisposing factors, but it was age groups, right? And I gave you specifically those bacteria. You’ll notice that in that second column, that there is no tuberculosis there because you’re keeping it separate. If you do that for me clinically, you’ll be in good shape. Now, the onset here will be insidious. Your typical symptoms that you would expect, photophobia, headaches, so on and so forth. And here, it causes a? Please focus on basilar meningitis. In other words, secondary angiitis could also be possible because of its location. Is that clear? The most important point in this slide, in this section, is going to be that last statement of where it’s affecting your meninges. And what you’re finding here on your CT, now granted, in your particular point in education that by looking at the CT, you would need to have proper history so that you would then identify what you find in that arrow as being the actual pathology. Let’s continue. Complications: They may result in hydrocephalus, but for the most part, whenever you have meningitis, at some point in time, you’re going to scar. You could scar the arachnoid granulation. Picture that for me. Close your eyes. Are you with me? Arachnoid granulation, where are you? Subarachnoid space and this will be the drainage point of your cerebrospinal fluid. What if you scar them? If you scar them and if you’re not familiar with the...

    About the Lecture

    The lecture Tuberculous Meningitis by Carlo Raj, MD is from the course CNS Infection—Clinical Neurology.


    Included Quiz Questions

    1. Basal region
    2. Corpus callosum
    3. Frontal cortex
    4. Pituitary gland
    5. Occipital cortex
    1. Steroids
    2. Surgical decompression
    3. Rifampicin
    4. Moxifloxacin
    5. Ventriculostomy
    1. Hydrocephalus
    2. Subarachnoid hemorrhage
    3. Meningioma
    4. Pseudocyst
    5. Calcification
    1. Due to vasculitis of the end arteries to the brain.
    2. Due to colonies of mycobacteria obstructing the end arteries of the brain.
    3. Due to rupture of the end arteries of the brain.
    4. Due to increase in neutrophils causing obstruction to the end arteries of the brain.
    5. Due to atherosclerosis of the end arteries of the brain.

    Author of lecture Tuberculous Meningitis

     Carlo Raj, MD

    Carlo Raj, MD


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