Lectures

Thyroiditis – Hyperthyroidism (Thyrotoxicosis)

by Carlo Raj, MD
(1)

Questions about the lecture
My Notes
  • Required.
Save Cancel
    Learning Material 2
    • PDF
      Slides HyperthyroidismGravesDisease EndocrinePathology.pdf
    • PDF
      Download Lecture Overview
    Report mistake
    Transcript
    Let us now move on to thyroiditis with hyperthyroidism. We’ve officially completed our discussion of Grave’s. Let’s get into the group of diseases characterized by thyroid inflammation. Chronic lymphocytic, the most common... Now, this is interesting. Hashimoto. Hashimoto, most common cause of primary hypothyroidism in the US. Hypothyroidism. So, why is it under hyperthyroidism? Because of the following. Whenever you have inflammation, -itis, inflammation of the thyroid, there’s every possibility that the colloid… Can you picture the colloid for me? The homogenous eosinophilic central region of your thyroid cell that stores your, harbours my thyroid hormones. Any time there’s inflammation, that colloid “explodes”. You may then release your T3, T4 into circulation in great quantity. Welcome to thyroid storm. There’s every possibility with any one of these thyroiditises that the patient may present initially with hyperthyroidism and that’s the topic that we’re sticking with here. Do not ever forget that. Chronic lymphocytic thyroiditis. Acute bacterial (suppurative) thyroiditis – rare, but still possible. Subacute thyroiditis. What’s subacute mean? Slow, slow. Subacute thyroiditis. De Quervain, granulomatous thyroiditis. Once again, destruction release of… Colloid releasing T3, T4 excessively. Silent thyroiditis. We’ll talk about this post-partum especially, with delivery. We’ll see that coming up. Amiodarone. Amiodarone. Take a look at the “iod” in amiodarone. And doesn’t that resemble iodine? With amiodarone… You may be using this as an antiarrhythmic drug specifically and technically comes under class 3. However, you know it has all classes of antiarrhythmic action. Point is, the iodine, with amiodarone, some patients will respond with hyperthyroid type of presentation, whereas other patients may present with hypo. I wish to be very clear here that the functioning or the side effect of amiodarone could either be hypo or hyper. You really want to pay attention how is your patient presenting. And we...

    About the Lecture

    The lecture Thyroiditis – Hyperthyroidism (Thyrotoxicosis) by Carlo Raj, MD is from the course Thyroid Gland Disorders.


    Included Quiz Questions

    1. Silent thyroiditis
    2. DeQuervain's thyroiditis
    3. Acute bacterial thyroiditis
    4. Riedel's thyroiditis
    5. Amiodarone induced thyroiditis
    1. Fistula from piriform sinus
    2. Underlying TB infection
    3. Hematogenous spread
    4. Aspergillus
    5. Pneumocystis
    1. de Quervains thyroditis
    2. Suppurative thyroditis
    3. Amiodarone induced thyroiditis
    4. Silent thyroiditis
    5. Riedel's thyroiditis
    1. Treatment includes IV antibiotics
    2. HLA-B35
    3. Mumps and coxsackie viruses
    4. A period of thyrotoxicosis followed by hypothyroidism
    5. Focal destruction of thyroid tissue
    1. Higher instance of elevated levels of serum antithyroid antibody
    2. Diffuse goiter or no thyroid enlargement
    3. Autoimmune etiology
    4. Mild hyperthyroidism
    5. Painless
    1. Glucocorticoids
    2. Pregnancy
    3. IFN-alpha
    4. IL-2
    5. Lithium

    Author of lecture Thyroiditis – Hyperthyroidism (Thyrotoxicosis)

     Carlo Raj, MD

    Carlo Raj, MD


    Customer reviews

    (1)
    5,0 of 5 stars
    5 Stars
    5
    4 Stars
    0
    3 Stars
    0
    2 Stars
    0
    1  Star
    0