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Benign Rolandic Epilepsy (BCECTS)

by Carlo Raj, MD
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    Let’s talk about epilepsy syndromes. Benign rolandic epilepsy. Age of onset: Childhood. Seizure types: Simple. What does partial mean to you? How much of the brain? Focal, involving mouth and face. It could be also generalized tonic-clonic. What does that mean to you? The entire brain. And how is your patient going to present? All four extremities, extended, and then clonic jerking movements. Associated findings: Nocturnal preponderance of seizures. Nocturnal, at night. EEG finding: We have temporal spikes or centrotemporal spikes Treatment: Frequently, no treatment is needed as seizures are nocturnal and self-limited. Maybe, perhaps you’re thinking about a sodium channel blocker known as carbamezapine. We’ll get into treatment further, but at this point, benign rolandic epilepsy. Luckily, most of the time, with this being a nocturnal issue, self-limited in nature. Contratemporal spikes is the clinical pearl. Here, it’s called Lennox Gastaut syndrome. Huh, interestingly enough, there’s been this huge push. Well, not just push, but let’s just say, cannabis has taken on quite a bit of media attention, hasn’t it? CNN, all of these, you know about Dr. Gupta and his studies and his investigations on children that have had a particular epileptic issues such as Lennox Gastaut syndrome. Let’s begin. Age of onset: Childhood, early adolescence. I’m going to be very deliberate here, because this condition is no joke. Seizure type: Tonic-Clonic, myoclonic, generalized. All over the place, huh? All over the place. What else? Mental retardation association. EEG findings -- I need you to know this -- slow, 1 to 2 hertz, paroxysmal fast activity, multifocal spikes. Now, what you need to know at this point for management is not going to be cannabis, that is not going to be an answer. Please don’t choose that. Anyhow, the point is we know that marijuana and cannabis has...

    About the Lecture

    The lecture Benign Rolandic Epilepsy (BCECTS) by Carlo Raj, MD is from the course Seizures. It contains the following chapters:

    • Benign Rolandic Epilepsy
    • Lennox-Gastaut Syndrome
    • Absence Epilepsy
    • Juvenile Myoclonic Epilepsy
    • Adult Epilepsy

    Included Quiz Questions

    1. Absence epilepsy
    2. Lennox-Gastaut syndrome
    3. Benign Rolandic epilepsy
    4. Juvenile myoclonic epilepsy
    5. Adult epilepsy
    1. Benign Rolandic seizures
    2. Absence seizures
    3. Lennox-Gaustaut Syndrome
    4. Juvenile myoclonic epilepsy
    5. Adult epilepsy
    1. Benign Rolandic Epilepsy shows centrotemporal spikes on EEG.
    2. Absence seizures show 4 Hz spikes on EEG.
    3. Lennox-Gestaut show 4 - 6 Hz spike and wave pattern on EEG.
    4. Adult epilepsy show slow (1 - 2Hz) spikes on EEG.
    5. Juvenile myoclonic epilepsy show 3 Hz spikes on EEG
    1. Lamotrigine
    2. Controlled Marijuana drug
    3. Ethosuximide
    4. Carbamazepine
    5. Benzodiazepine
    1. Temporal sclerosis
    2. Frontal sclerosis
    3. Parietal sclerosis
    4. Occipital sclerosis
    5. Fronto-temporal sclerosis

    Author of lecture Benign Rolandic Epilepsy (BCECTS)

     Carlo Raj, MD

    Carlo Raj, MD


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