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Acute and Chronic Muscle Spasms – Skeletal Muscle Relaxants

by Pravin Shukle, MD
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    About the Lecture

    The lecture Acute and Chronic Muscle Spasms – Skeletal Muscle Relaxants by Pravin Shukle, MD is from the course CNS - Pharmacology. It contains the following chapters:

    • Chronic Muscle Spasm Agents
    • Dantrolene

    Included Quiz Questions

    1. Brainstem
    2. Peripheral muscles
    3. Pons
    4. Cerebellum
    5. Primary nerves of the spine
    1. Diazepam interacts with the GABA-A receptor to increase frequency of channel opening.
    2. Diazepam interacts with the GABA-B receptor to inhibit the G protein coupled receptor (GPCR).
    3. Diazepam interacts with the GABA-B receptor to inhibit the adenylyl cyclase.
    4. Diazepam interacts with the GABA-A receptor to increase duration of channel opening.
    5. Diazepam forces ion channels open resulting in a polarization of the muscle and then depolarization of the muscle and relaxation.
    1. A 65-yea-old male with history of muscle spasticity after suffering an ischemic stroke.
    2. A 74-yea-old male with a history of chronic back pain and metastatic prostate cancer.
    3. A 35-yea-old male with a history of ankylosing spondylitis and chronic lower back pain.
    4. A 24-yea-old female with a history of opioid and benzodiazepine dependency complaining of vague muscle pains throughout her body and demanding immediate treatment.
    5. A 67-yea-old female with a history of shuffling gait, pill rolling tremor and low grade dementia.
    1. Cyclobenzaprine: nicotinic-acetylcholine receptor
    2. Cyclobenzaprine: muscarinic-acetylcholine receptor
    3. Baclofen: GABA-B receptor
    4. Diazepine: GABA-A receptor
    5. Tizanidine: alpha-2 receptors

    Author of lecture Acute and Chronic Muscle Spasms – Skeletal Muscle Relaxants

     Pravin Shukle, MD

    Pravin Shukle, MD


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