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Rapidly Progressive Glomerulonephritis (rpgn) & Subtypes

by Carlo Raj, MD
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    The lecture Rapidly Progressive Glomerulonephritis (rpgn) & Subtypes by Carlo Raj, MD is from the course Glomerulonephritis.


    Included Quiz Questions

    1. Progression to acute renal failure in 4-12 weeks
    2. Proliferation of visceral epithelial cells
    3. Tubular dysfunction
    4. Chronic renal failure
    5. Clinical syndrome that must be the primary type of glomerular disease
    1. Parietal epithelial cell
    2. Visceral epithelial cell
    3. Fenestrated cells
    4. Endothelial cell
    5. Mesangial cell
    1. Minimal change disease
    2. Post-streptococcal glomerulonephritis
    3. Goodpasture syndrome
    4. Granulomatosis with polyangiitis
    5. IgA nephropathy
    1. It represents anti-GBM disease
    2. It presents with granular deposits on immunofluorescence
    3. It most commonly affects the elderly
    4. It may be diagnosed using electron microscopy
    5. It involves IgM deposition on the GBM
    1. Fibrin crescents with H&E stain on light microscopy
    2. Negative findings on immunofluorescence
    3. Polyuria
    4. Proteinuria > 3.5 g/day
    5. Lack of RBC casts in the urine
    1. Henoch-Schonlein purpura associated type 2 RPGN involves IgM complex deposits
    2. SLE-associated type 2 RPGN involves subendothelial deposits
    3. Infectious etiologies for type 2 RPGN usually involve subepithelial deposits
    4. Type 2 RPGN involves deposition of preformed immune complexes
    5. All of the above are correct
    1. Anti-myeloperoxidase
    2. Anti-proteinase 3
    3. Anti-dsDNA
    4. Anti-saccharomyces cerevisiae
    5. Cytoplasmic antineutrophil cytoplasmic antibodies

    Author of lecture Rapidly Progressive Glomerulonephritis (rpgn) & Subtypes

     Carlo Raj, MD

    Carlo Raj, MD


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