by Carlo Raj, MD

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    We’ll continue our discussion of dermatology by looking at angioedema. Take a look at these lovely lips. Well, this was not implantation. This was not a patient that went in for cosmetic surgery and had it enhanced. So, what do you think happened here? Well, there are two ways in which your patient may develop what’s known as angioedema. To begin with, by definition, when you have angioedema, it is the fact that you have edema that’s taking place. Obviously, it’s leaving, or should I say the fluid is escaping from the angio, which refers to your blood vessels. And when it does, in terms of its location, most likely, it would be deep into your skin, so you’re thinking about dermis, and maybe perhaps even, your subcutaneous tissue. With that said, our topic is angioedema. It could be acquired or hereditary. And with skin, the last time we talked about this was, in fact, a type 1 hypersensitivity in which you would be releasing histamine, and that would be a type of acquired angioedema. And at some point in time, we’ve also discussed hereditary type of angioedema, which from immunology, will be more or less considered to be a complement type of pathology, in which your patient is then deficient of C1-inhibitor. All those should come in mind, and we have swelling of the lips, airways, or eyes when dealing with angioedema The causes include -- well, in order for you to understand this, it’s the concept that you want to keep in mind. The concept here is going to be the fact that you are not able to properly control with hereditary. Your complement system. And by this, we mean, for example, we’ve talked about a few complement pathologies, in which you have unrestricted or unopposed complement activity....

    About the Lecture

    The lecture Angioedema by Carlo Raj, MD is from the course Inflammatory Skin Diseases.

    Included Quiz Questions

    1. Urticaria or hives
    2. Swelling of lips and tongue
    3. Low C4 levels
    4. C1 esterase inhibitor deficiency
    5. Unopposed complement activity
    1. Stop Lisinopril
    2. Stop Amlodipine
    3. Continue current regimen and add antihistamines
    4. Add IV steroids
    5. Continue current treatment and transfuse FFP
    1. contact dermatitis
    2. PNH (paraoxysmal nocturnal hematuria)
    3. MPGN Type 2
    4. Hereditary angioedema
    5. Dense deposit disease

    Author of lecture Angioedema

     Carlo Raj, MD

    Carlo Raj, MD

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