Lectures

Gastrointestinal System Question Set 2

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    About the Lecture

    The lecture Gastrointestinal System Question Set 2 by Lecturio USMLE is from the course Gastrointestinal System – Board-Style Questions.


    Included Quiz Questions

    1. Non tropical Sprue
    2. Tropical sprue
    3. Whipple’s Disease
    4. Small Intestinal bacterial overgrowth
    5. Irritable bowel disease
    1. Amoebiasis
    2. Ulcerative colitis
    3. Crohns disease
    4. Diverticulosis
    5. Salmonellosis
    1. Hemochromatosis
    2. Psychogenic erectile dysfunction
    3. Wilsons disease
    4. Chronic hepatitis
    5. Porphyria cutanea tarda
    1. DNA mismatch repair
    2. Inhibitor of apoptosis
    3. Cytoskeletal stability
    4. RAS cycle transduction inhibitor
    5. Inhibits progression from G1 to S phase
    1. Upper esophageal web
    2. Esophageal carcinoma
    3. Lower esophageal spasm
    4. Lower esophageal ring
    5. Failure of the relaxation of lower esophageal sphincter
    1. Disturbed intestinal motility
    2. Lactase deficiency
    3. Lack of nerve plexus in submucosa
    4. Ischemia to Colon
    1. Decrease in beta oxidation
    2. Increase in glycolysis
    3. Increase in beta Oxidation
    4. Increase in gluconeogenesis
    5. Decrease in electron transport chain
    1. CD4+ cell
    2. NK cell
    3. CD14+ cell
    4. CD19+ cell
    1. Cytoplasmic fatty vacuolization in hepatocytes and swollen mitochondria
    2. Iron accumulation and proliferation of Smooth endoplasmic reticulum.
    3. Hepatic granulomas
    4. Bridging hepatic necrosis and fibrosis
    5. Hepatocytes proliferation
    1. Diverticulitis
    2. Chronic inflammatory bowel disease
    3. Cancer of the colon
    4. Hypothyroidism
    5. Irritable bowel syndrome
    1. Anti SS-B antibody
    2. Anti double stranded DNA
    3. Anti centromere antibody
    4. Anti -Scl 70 antibody
    5. Anti -Jo-1 antibody
    1. Mucosal atrophy
    2. Increased number of intraepithelial lymphocytes
    3. Crypt hyperplasia
    4. Reduced height of villi
    5. Cuboidal appearance of surface epithelial cells
    1. Gilbert syndrome
    2. Physiological jaundice
    3. Crigler – Najjar syndrome type I
    4. Crigler – Najjar syndrome type II
    5. Hemolytic anemia
    1. Crigler – Najjar syndrome type I
    2. Gilbert syndrome
    3. Neonatal jaundice
    4. Crigler – Najjar syndrome type II
    5. Hemolytic anemia.
    1. α1-antitrypsin deficiency
    2. Dubin – Johnson syndrome
    3. Gilbert syndrome
    4. Glucose-6-phosphate dehydrogenase deficiency
    5. Hepatitis C
    1. Primary biliary cirrhosis
    2. Hepatitis A
    3. Hemolytic anemia
    4. Crigler – Najjar syndrome type I
    5. Gilbert syndrome
    1. Primary sclerosing cholangitis
    2. Hepatitis A
    3. Hepatitis B
    4. Primary biliary cirrhosis
    5. Hepatitis E
    1. No therapy indicated
    2. Phenobarbital
    3. Phototherapy
    4. Plasma exchange transfusion
    5. Inhibitors of heme oxygenase
    1. Plasma exchange transfusion
    2. Phenobarbital
    3. Continuation of phototherapy
    4. Furosemide
    5. No therapy
    1. Autosomal recessive
    2. Autosomal dominant
    3. X-linked
    4. Y-linked
    5. Codominant
    1. Endoscopic retrograde cholangiopancreatography (ERCP)
    2. Echosonography of the abdomen
    3. CT of the abdomen
    4. Liver biopsy
    5. Magnetic resonance cholangiopancreatography
    1. Onion skin fibrosis (concentric periductal fibrosis)
    2. Mononuclear cell infiltrate and apoptotic hepatocytes
    3. Damage of the basement membrane of the biliary ducts and reactive hyperplasia of the epithelial lining
    4. Small drops of fat all over the liver tissue
    5. Normal finding
    1. Ischemic colitis
    2. Pseudomembranous colitis
    3. Appendicitis
    4. Crohn’s disease
    5. Perforated duodenal ulcer
    1. Paracentesis
    2. Esophagastroduodenoscopy
    3. CT scan of the head
    4. Serum ammonia level
    5. Therapeutic trial of lactulose
    1. Drotrecogin alfa
    2. Surgical consultation
    3. Broad-spectrum antibiotics
    4. Abdominal radiograph
    5. Intravenous fluid
    1. ERCP – Endoscopic retrograde cholangiopancreatography
    2. HIDA scan
    3. Antibiotics and observation
    4. Hepatitis serologies
    5. Serologies for antimitochondrial antibodies
    1. Gilbert’s syndrome
    2. Cholelithiasis
    3. Crigler-Najjar syndrome type 1
    4. Dubin-Johnson syndrome
    5. Medication-induced hemolysis
    1. Budd-Chiari syndrome
    2. Acute hepatitis B infection
    3. Acute hepatitis A infection
    4. Acute hepatitis C infection
    5. Acetaminophen ingestion
    1. Forced cough elicits abdominal pain.
    2. Hyperactive bowel sounds are heard on auscultation.
    3. Bowel sounds are not observed on auscultation.
    4. Rectal examination shows hemepositive stools
    5. Pain is arousing with gentle intensity/pressure at the costo-vertebral angle.
    1. Adenovirus
    2. Influenza virus
    3. Enterovirus
    4. Metapneumovirus
    5. Rhinovirus
    1. Gram positive, branching rod
    2. Gram positive cocci in clusters
    3. Enveloped, double stranded DNA virus
    4. Gram positive cocci in chains
    5. Aerobic gram negative rod
    1. This lesion is non-contagious but will most likely recur.
    2. This lesion is highly contagious and is due to reactivation of a dormant virus.
    3. This lesion is due to a fungal infection and may mean you’re immunocompromised.
    4. This lesion may progress to squamous cell carcinoma.
    5. This lesion is associated with an autoimmune disease characterized by sensitivity to gluten.
    1. Maternal iodine deficiency
    2. Excess growth hormone secondary to pituitary gland tumor
    3. Mutation in the WT2 gene
    4. Type I hypersensitivity reaction
    5. Autosomal dominant mutation in the SERPING1 gene
    1. Pernicious anemia
    2. Plummer-Vinson syndrome
    3. Kawasaki disease
    4. Herpes simplex virus-1 infection
    5. Oral candidiasis infection
    1. This lesion necessitates biopsy.
    2. This lesion carries no increased risk of cancer.
    3. This lesion is due to an infection
    4. Tobacco use is not a risk factor for this.
    5. This lesion is closely associated with chronic Hepatitis C infection.
    1. Benign salivary gland tumor composed of stromal and epithelial tissue
    2. An infection with Paramyxovirus
    3. Benign cystic tumor with stroma resembling lymph node tissue
    4. Sialolithiasis that has progressed to a S. aureus infection
    5. Malignant tumor composed of squamous and mucinous cells
    1. Scleroderma
    2. Diffuse esophageal spasm
    3. Zenker’s diverticulum
    4. Achalasia
    5. Polyomyositis
    1. Increased pressure in the distal esophageal vein due to increased pressure in the left gastric vein
    2. Perforation of the gastric mucosa
    3. Lacerations of the mucosa at the gastroesophageal junction
    4. Inflammation of the portal tract due to a chronic viral illness
    5. Decreased GABA activity due to downregulation of receptors
    1. Increased pressure above the upper esophageal sphincter resulting in a defect in the wall
    2. Outpouching of all 3 layers of the esophageal mucosal tissue distal to the upper esophageal sphincter
    3. Failure of neural crest migration into the Auerbach plexus
    4. Persistence of an embryologic structure
    5. Inability to relax the lower esophageal sphincter
    1. Protrusion of fundus of the stomach through the diaphragm into the thoracic cavity
    2. “Hourglass stomach” due to upward displacement of the gastroesophageal junction
    3. Lung hypoplasia due to a defect in the diaphragm
    4. Cardiomegaly with pulmonary effusion
    5. Widened mediastinum with evidence of esophageal rupture
    1. Decreased lower esophageal sphincter tone
    2. Blockage of the cystic duct leading to inflammation of the wall of the gallbladder
    3. Autodigestion of pancreatic tissue
    4. Erosion of the mucosa of the antrum of the stomach
    5. An atherosclerotic blockage of a coronary artery causing transient ischemia during times of increased cardiac demand
    1. Metaplasia of the esophageal mucosa
    2. Longitudinal lacerations of the esophageal mucosa
    3. Hypertrophy of the esophageal mucosa protruding into the lumen of the lower esophagus
    4. A malignant proliferation of squamous cells
    5. Esophageal smooth muscle atrophy
    1. Candida albicans
    2. Cytomegalovirus
    3. Human papilloma virus
    4. Irritation due to medication therapy
    5. HHV-8
    1. Mallory-Weiss tear
    2. Boerhaave syndrome
    3. Gastric ulcer
    4. Hiatal hernia
    5. Esophageal varices
    1. Malignant proliferation of squamous cells
    2. Reduced lower esophageal pressure
    3. Transformation leading to metaplasia in the lower esophagus
    4. Malignant proliferation of glandular tissue in the esophagus
    5. Chronic autoimmune gastritis
    1. Adenocarcinoma
    2. Achalasia
    3. Gastric ulcers
    4. Squamous cell carcinoma
    5. Benign stricture
    1. Correct electrolyte imbalance
    2. Reassurance and observation
    3. Pyloromyotomy
    4. Whipple’s procedure
    5. Nasogastric tube feeding
    1. Cisapride
    2. Domperidone
    3. Erythromycin
    4. Metoclopramide
    5. Bethanechol
    1. Perform a colonoscopy at the age of 40 and repeat every five years.
    2. Perform a colonoscopy at the age of 40 and repeat every three years.
    3. Perform a colonoscopy now and repeat every ten years.
    4. Perform a colonoscopy at the age of 50 and repeat every five years.
    5. Perform a colonoscopy at the age of 50 and repeat every ten years.
    1. Descending colon
    2. Small bowel
    3. Cecum
    4. Ascending colon
    5. Rectosigmoid colo
    1. Luminal obstruction due to an enlarged lymphoid follicle
    2. Luminal obstruction preventing passage of gastrointestinal contents
    3. Twisting of testes on its axis hampering its blood supply
    4. Immune-mediated vasculitis associated with IgA deposition
    5. Ascending infection of the urinary tract
    1. Mutation in the TCOF1 gene
    2. Trisomy 18
    3. Retinoic acid use during gestation
    4. Microdeletion at chromosome 22q14
    5. Mutation of the SOX9 gene
    1. Lemierre syndrome
    2. Whooping cough
    3. Infectious mononucleosis
    4. Diphtheria
    5. Acute rheumatic fever
    1. Preventable by a live attenuated vaccine
    2. Is a common cause of septic arthritis in this patient’s age group
    3. Original presentation in the form of a painless chancre
    4. Cause buboes in the inguinal lymph nodes
    5. Dilation of the pampiniform plexus
    1. Coxsackievirus A
    2. Staphylococcus aureus
    3. Herpes simplex virus type 1
    4. Type II sensitivity reaction
    5. Varicella zoster
    1. A naked, double stranded DNA virus
    2. An enveloped, single stranded, negative sense RNA virus
    3. An enveloped, double stranded DNA virus
    4. A gram positive, beta-hemolytic cocci in chains
    5. A gram negative, pleomorphic, obligate intracellular bacteria
    1. Pylephlebitis
    2. Intestinal obstruction
    3. Appendiceal abscess
    4. Peritonitis
    5. Perforation
    1. Hemorrhoids
    2. Anal fissure
    3. Anorectal fistula
    4. Rectal ulcer
    5. Proctitis
    1. Hirschsprung disease
    2. Cystic fibrosis
    3. Enterobiasis
    4. Malnutrition
    5. Myelomeningocele
    1. Local anal trauma
    2. Inflammatory bowel disease
    3. Anorectal abscess
    4. Rectal prolapse and paradoxical contraction of the puborectalis muscle
    5. Deterioration of the connective tissue that anchors hemorrhoids
    1. Anal cancer
    2. Hemorrhoids
    3. Polyps
    4. Proctitis
    5. Anal fissure
    1. Fecal-oral
    2. Blood transfusion
    3. Sexual contact
    4. Breast milk
    5. Perinatal
    1. Viral hepatitis B
    2. Viral hepatitis A
    3. Viral hepatitis C
    4. Viral hepatitis D
    5. Viral hepatitis E
    1. Acute hepatitis B
    2. Chronic hepatitis B
    3. Past hepatitis B infection
    4. Acute hepatitis D superinfection
    5. Acute hepatitis A
    1. Ascending cholangitis
    2. Cholecystitis
    3. Pancreatitis
    4. Appendicitis
    5. Liver abscess
    1. Autoimmune hepatitis
    2. Primary biliary cholangitis
    3. Primary sclerosing cholangitis
    4. Secondary biliary cirrhosis
    5. Alpha-1 antitrypsin deficiency
    1. Idiopathic inflammatory scarring of bile duct
    2. Abnormal cystic dilation of biliary tree
    3. Liver fluke induced inflammation leading to metaplastic change
    4. Long term carcinogenic effect of contrast agent
    5. Chronic infection due to hepatitis virus
    1. Entrapped epithelial crypts seen as pockets of epithelium in the wall of the gallbladder
    2. Hyalinized collagen and dystrophic calcification in submucosal layer
    3. Minimal lymphoid aggregates
    4. Neutrophilic infiltration with vascular congestion and fibrin deposition in gall bladder
    5. Abnormal deposits of cholesterol ester in macrophages in lamina propria
    1. Bedside Index of Severity in Acute Pancreatitis (BISAP) scoring
    2. Ranson’s criteria
    3. Modified Glasgow scoring
    4. Acute Physiology and Chronic Health Examination (APACHE) II scoring
    5. C- reactive protein
    1. CA 19-9 is a marker for this condition.
    2. Patients with this condition often rapidly develop glucose intolerance and severe diabetes.
    3. The majority of cases are in the body of the pancreas.
    4. This condition is most common in caucasians.
    5. Caffeine consumption is an established risk factor for this condition.
    1. Anti HIV drug
    2. Gallstones
    3. Anti-diabetic medication
    4. Alcohol
    5. Abdominal trauma
    1. Turners syndrome
    2. Down’s syndrome
    3. Kartagener's syndrome
    4. Friedreich ataxia
    5. Marfan’s syndrome
    1. Decreased production of PGE2
    2. Increased stimulation of the vagus nerve
    3. Direct irritation of the stomach lining causing inflammation
    4. Hypovolemia
    5. Buildup of a toxic metabolite due to kidney disease
    1. Destruction of the mucosa of the stomach is mediated by T cells
    2. Serum gastrin levels are decreased
    3. MALT lymphoma is a common complication
    4. Caused by a gram negative rod that is urease positive
    5. It is the most common cause of folate deficiency in the US
    1. A urease positive organism
    2. The presence of a large amount of neutrophils with limited macrophages
    3. An abundance of self reactive T cells
    4. Absence or evidence of destruction of parietal cells
    5. Signet ring cells
    1. Serology (ELISA testing)
    2. Steiner’s stain
    3. Detection of the breakdown products of urea in biopsy
    4. Stool antigen test
    5. Culture of organisms from gastric specimen
    1. Zollinger Ellison syndrome
    2. H. pylori infection
    3. Intestinal type gastric adenocarcinoma
    4. Whipple disease
    5. NSAID induced peptic ulcer disease
    1. Chronic NSAID use
    2. Longstanding GERD
    3. Hypertension
    4. Adverse effect of beta blockers
    5. Age and gender
    1. Signet ring cells
    2. Ectopic thyroid tissue
    3. Intestinal metaplasia in the stomach
    4. Hyperplasia of gastric mucosa
    5. PAS positive macrophages
    1. Sigmoid colon
    2. Duodenum
    3. Ascending colon
    4. Stomach
    5. Ileum
    1. MEN 1
    2. Gastric adenocarcinoma, intestinal type
    3. VIPoma
    4. H. pylori infection
    5. Gastroesophageal reflux disease
    1. History of blood transfusion
    2. History of Hepatitis E
    3. Shortness of breathing
    4. History of alcoholism
    5. Hemochromatosis
    1. Budd-chiari syndrome
    2. Viral hepatitis
    3. Nonalcoholic fatty liver disease
    4. Hemochromatosis
    5. Drug-induced hepatitis
    1. Mallory-Denk bodies
    2. Steatosis alone
    3. “Florid” bile duct lesion
    4. Hürthle cells
    5. Gaucher cells
    1. Primary biliary cholangitis
    2. Choledocolithiasis
    3. Pancreatic cancer
    4. Hepatic amyloidosis
    5. Fasciolasis
    1. Alcoholic liver disease
    2. Chronic viral hepatitis
    3. Hemochromatosis
    4. Non alcoholic fatty liver disease
    5. Hepatic adenoma
    1. Spontaneous bacterial peritonitis
    2. Variceal gastrointestinal bleeding
    3. Portal vein thrombosis
    4. Metabolic alkalosis
    5. Hypoglycemia
    1. Portal hypertension
    2. Congestive gastropathy
    3. Hepatorenal syndrome
    4. Spontaneous bacterial peritonitis
    5. Hepatocellular carcinoma
    1. Secretory diarrhea
    2. Invasive diarrhea
    3. Osmotic diarrhea
    4. Steatorrhea
    5. Motility diarrhea
    1. Pancreatic insufficiency
    2. Chronic damage to intestinal mucosa
    3. Damage to intestinal brush border
    4. Autoimmune damage to parietal cells
    5. Decreased recycling of bile acids
    1. Fecal fat test
    2. Stool guaiac test
    3. Colonoscopy
    4. CT with oral contrast
    5. Stool O&P
    1. Anti-tTG or gliadin antibodies
    2. Anti-histone antibodies
    3. Anti-lactase antibodies
    4. HLA-B27
    5. HLA-DQ2
    1. PAS (+) macrophages
    2. Blunting of the microvilli
    3. Signet ring cells
    4. A gram (-), urease (+) helical organism
    5. Non-caseating granulomas in the small intestine
    1. Mass effect from a tumor
    2. Crohn’s disease
    3. Incarcerated hernia
    4. Volvulus at the splenic flexure of the colon
    5. Adhesions
    1. Medications
    2. Crohn’s disease with ileitis
    3. Lymphocytic colitis
    4. Lactose intolerance
    5. Carcinoid tumor
    1. Cryptosporidia
    2. Campylobacter
    3. Entamoeba
    4. Escherichia coli
    5. Shigella
    1. Calcium
    2. Glucose
    3. Lipase
    4. Trypsinogen
    5. C-reactive protein

    Author of lecture Gastrointestinal System Question Set 2

     Lecturio USMLE

    Lecturio USMLE


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