00:01
If we were to look at
the valves all together
in a single view with
the atria removed,
we can see just how similar right
and left valves look to each other.
00:10
The pulmonary valve that's sitting
just anterior to the aortic valve
looks so similar.
00:16
And that's actually because
they came from the same artery
long time ago during development,
and just divided into two almost
mirror images of each other.
00:25
We call these semilunar valves
because they're basically
little halfmoon shapes.
00:30
And they don't have any chordae
tendineae or papillary muscles.
00:33
They kind of closed
by passive closure.
00:36
But the mitral valve
and the tricuspid valve,
were quite a bit larger.
00:40
Again, very similar except for the
number of leaflets that they had.
00:44
And these are called
atrioventricular valves,
because they're between
the atria and the ventricles.
00:51
So those atrioventricular
valves have a bit more going on.
00:55
And it makes sense
because they're valves
that are going to prevent backflow
coming from the ventricle
and the ventricles are
pumping really hard.
01:03
And so in order to prevent blood
from getting shot back
into the atria,
you're going to need a
lot stronger structure
than you will from passive flow
from a pulmonary artery or aorta.
01:16
So here we see the cusps of
those atrial ventricular valves.
01:19
Again, attached to papillary
muscles via chordae tendineae.
01:24
And you can see as
the heart's beating,
how they go into action?
So the pulmonary and
aortic valve structures
have really nothing
attached to them.
01:34
They don't really need to, they're
shaped as everything you need.
01:37
They're convex, or sort of curved
on the ventricle side,
so blood can just
push them through.
01:43
And then they're cupped on
the other side are concave,
so that when blood tries to
come back, they fill these cusp,
and it closes them off,
and you don't get any backflow
leaking backwards
back into the ventricles.
01:56
So let's look at the valves
during ventricular contraction,
also known as systole.
02:02
So when the myocardium,
or heart muscle is contracted,
we see that blood is being
pushed through the aortic valve.
02:09
It's open, we say.
02:11
But we don't want blood going
backwards into the atrium,
so the mitral valve is closed.
02:16
You can see where these chordae
tendineae and papillary muscles
are holding tight to make sure
it doesn't get forced
backwards into the atrium.
02:27
During ventricular relaxation,
also known as diastole,
we have the opposite,
the myocardium is relaxed.
02:34
and it's going to fill now with
blood coming from the atrium.
02:39
But we only want blood
coming from the atrium,
meaning blood trying to come
backwards from the aorta
gets closed off
by the aortic valve, passively.
02:49
And then the mitral valve is opened,
so that that atrial blood
can go through
and fill the ventricle and
start the cycle over again.
02:59
And this all assumes that the valves
are working as they're supposed to.
03:03
Meaning the valves are thin,
they're compliant,
they move out of the way
when they're supposed to, and
they close when they're supposed to.
03:10
Unfortunately, because valves are
opening, closing under pressure,
all the time, they accumulate
a lot of wear and tear.
03:18
And eventually they can become
calcified, and very rigid.
03:22
And when that happens,
it can't open very well.
03:26
And when anything along
the circulatory system
can't open very well,
we say it's stenotic.
03:31
So this is a very common
condition called aortic stenosis,
that's going to make
it really hard to open
during systole or ventricular
contraction, the aortic valve.
03:44
So under normal conditions,
the left ventricle
is going to contract
just enough to make sure
that the systemic circulation
is going to receive enough pressure
to perfuse all the arteries
that our body needs.
03:57
But if the aortic
valve becomes stenotic,
that's going to put a lot
more stress and strain
on the left ventricle,
because it's trying to force
its blood through a tinier opening.
04:08
And over time, just like any
muscle that's doing more work
than it's supposed to,
it's going to become hypertrophied.
04:15
And unfortunately,
if that goes for too long,
it can't compensate anymore,
and it can actually start to fail.
04:23
If we look in a bit greater detail
around the area of the aortic valve,
we notice some
interesting features.
04:29
So here's the very beginning
of our ascending aorta.
04:33
But before we even get to that
we see are these little holes
just above the leaflets
of the valve.
04:39
We see one on the right called
the right coronary ostium.
04:43
Ostium is just
another word for hole.
04:45
And that's going to be the opening
for our right coronary artery.
04:49
That is the artery that supplies
the right side of the heart.
04:52
Similarly,
we have a hole on the left
called the left coronary ostium.
04:57
That's going to be opening
for our left coronary artery.
05:01
The cusp in the middle
is actually our posterior cusp,
but it's also known as the
non-coronary cusp for this reason,
it doesn't supply
any coronary arteries.