Lectures

Hematology Question Set 2

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    About the Lecture

    The lecture Hematology Question Set 2 by Lecturio USMLE is from the course Hematology – Board-Style Questions.


    Included Quiz Questions

    1. Vitamin B12 deficiency
    2. Folic acid deficiency
    3. Iron deficiency
    4. Vitamin C deficiency
    5. Vitamin A deficiency
    1. Proper diet and iron supplementation
    2. Proper diet only
    3. Lead supplementation
    4. iron supplementation only
    5. Multivitamins
    1. Immune thrombocytopenic purpura (ITP)
    2. Von Willebrand disease
    3. Acute lymphoblastic leukemia (ALL)
    4. Hemophilia A
    5. Hemophilia B
    1. Coumadin is antagonizing vitamin K reductase necessary for production of factors II, VII, IX and X
    2. Local irritation of bowels because of the ingestion of rodenticide
    3. Coumadin is antagonizing vitamin K reductase necessary for production of factors VIII and IX
    4. Coumadin caused thrombocytopenia
    5. Coumadin caused disseminated intravascular coagulation (DIK)
    1. Hemolytic anemia caused by glucose-6-phosphate dehydrogenase deficiency
    2. Microcytic anemia caused by iron deficiency
    3. Anemia caused by renal failure
    4. Autoimmune hemolytic anemia
    5. Aplastic anemia
    1. Dog bite
    2. Human bite
    3. Cat bite
    4. Fish bite
    5. Spider bite
    1. Hemorrhage
    2. Peripheral neuropathy
    3. Deep venous thrombosis
    4. Night blindness
    5. Retinopathy
    1. Hemolytic anemia
    2. Leukopenia
    3. Drug-associated lupus
    4. Hyperthyroidism
    5. Rash
    1. Administration of intravenous immunoglobulins
    2. Continuation of corticosteroids
    3. Transplantation of the stem cells
    4. Splenectomy
    5. Transfusion of the thrombocytes
    1. Anemia of chronic disease
    2. Megaloblastic anemia
    3. Sideropenic anemia
    4. Thalassemia
    5. Hemolytic anemia
    1. Splenectomy
    2. Continuation of corticosteroids
    3. Transplantation of the stem cells
    4. Administration of intravenous immunoglobulins
    5. Transfusion of the thrombocytes
    1. Von Willebrand disease
    2. Hemophilia A
    3. Hemophilia B
    4. Acute lymphoblastic leukemia
    5. Immune thrombocytopenic purpura
    1. Pyrimidine 5’-nucleotidase deficiency
    2. Lead poisoning
    3. Glucose-6-phosphate dehydrogenase deficiency
    4. Pyruvate kinase deficiency
    5. Cytochrome b5 reductase deficiency
    1. Hepatitis C virus
    2. Epstein-Barr virus
    3. Human T-cell lymphotropic virus (HTLV)
    4. Human immunodeficiency virus (HIV)
    5. Human herpes virus 8
    1. Serum levels of fibrinogen in a premature infant born at 32-weeks of gestation are significantly increased as compared to an adult.
    2. An extremely premature infant has marked reduced levels of protein C as compared to an adult.
    3. There is physiologic reduction in levels of antithrombin III in neonates.
    4. A transient decrease in serum levels of factor VII is seen in almost all neonates, which returns to normal levels by 7-10 days of age.
    5. Administration of vitamin K1 to the mother during labor has no effect on incidence of widespread subcutaneous ecchymoses that may be seen immediately after birth in otherwise normal premature infants.
    1. t(9;22)
    2. Trisomy 4
    3. t(12;21)
    4. Hyperdiploidy
    5. Trisomy 10
    1. Acute leukemia
    2. Chronic leukemia
    3. Immunologic thrombocytopenic purpura
    4. Aplastic anemia
    5. Tuberculosis of the bone marrow
    1. Sideropenic anemia
    2. Lead poisoning
    3. Pernicious anemia
    4. Glucose-6-phosphate dehydrogenase deficiency
    5. Hemolytic anemia
    1. Acute lymphoblastic leukemia
    2. Chronic lymphocytic leukemia
    3. Chronic myeloid leukemia
    4. Aplastic anemia
    5. Acute myeloid leukemia
    1. Hemolytic anemia
    2. Sideropenic anemia
    3. Anemia of chronic disease
    4. Acute leukemia
    5. Aplastic anemia
    1. Hemoglobin: decreased, Hematocrit: decreased, Red blood cell count: decreased, Mean corpuscular volume: increased
    2. Hemoglobin: decreased, Hematocrit: decreased, Red blood cell count: increased, Mean corpuscular volume: increased
    3. Hemoglobin: decreased, Hematocrit: increased, Red blood cell count: decreased, Mean corpuscular volume: decreased
    4. Hemoglobin: increased, Hematocrit: increased, Red blood cell count: increased, Mean corpuscular volume: increased
    5. Hemoglobin: increased, Hematocrit: decreased, Red blood cell count: decreased, Mean corpuscular volume: increased
    1. Ferritin: increased, Total iron binding capacity: decreased, Serum iron: increased
    2. Ferritin: decreased, Total iron binding capacity: decreased, Serum iron: decreased
    3. Ferritin: decreased, Total iron binding capacity: increased, Serum iron: decreased
    4. Ferritin: normal, Total iron binding capacity: normal, Serum iron: normal
    5. Ferritin: increased, Total iron binding capacity: decreased, Serum iron: decreased
    1. GPIIb/IIIa
    2. Factor IX
    3. Factor XI
    4. Vitamin K
    5. Antithrombin III
    1. Fibrinogen
    2. Giant platelets
    3. GpIIb/IIIa
    4. COX-1 and COX-2
    5. Vitamin K
    1. X
    2. XII
    3. XIIa
    4. VII
    5. VIIa
    1. Tropical sprue
    2. Poor diet
    3. Celiac disease
    4. Autoimmune destruction of parietal cells
    5. Diphyllobothrium latum infection
    1. Fetal hemoglobin is elevated
    2. Occurs due to a defect in lymphoblasts and erythroid progenitor cells
    3. Occurs due to inability to convert orotic acid to Uridine Monophosphate (UMP)
    4. Occurs due to antibodies against parietal cells of the stomach
    5. Splenectomy is a treatment option
    1. Her lab work likely demonstrates a normocytic anemia
    2. Her reticulocyte count is expected to be lower than normal
    3. She is likely to be diagnosed with iron deficiency anemia
    4. Hemoglobin levels are expected to be low right after the accident
    5. Hematocrit is expected to be low right after the accident
    1. Aplastic crisis
    2. Anemia of chronic disease
    3. Reaction to the blood transfusions
    4. Sequestration crisis
    5. Gastrointestinal bleeding
    1. Missense mutation
    2. Silent mutation
    3. Nonsense mutation
    4. Frameshift mutation
    5. Splice site
    1. Precursor T acute lymphoblastic leukemia/lymphoma
    2. Hairy cell leukemia
    3. Classic Hodgkin’s lymphoma
    4. Precursor B acute lymphoblastic leukemia/lymphoma
    5. Diffuse large B cell lymphoma
    1. HTLV-1
    2. HIV
    3. Hepatitis B
    4. Hepatitis C
    5. HTLV-2
    1. Tartrate Resistant Acid Phosphatase (TRAP)
    2. Monospot
    3. Quantiferon Gold
    4. CD 25
    5. Rapid streptococcal antigen
    1. Localized painless lymphadenopathy
    2. Generalized painful lymphadenopathy
    3. Localized painful lymphadenopathy
    4. Generalized painless lymphadenopathy
    5. Reactive lymphadenitis
    1. Sentinel node – Right lumbar trunk – Cisterna chyli – Thoracic duct – Left subclavian vein – Systemic circulation
    2. Sentinel node – Right lumbar trunk – Cisterna chyli – Thoracic duct – Right subclavian vein – Systemic circulation
    3. Sentinel node – Left lumbar trunk – Cisterna chyli – Thoracic duct – Left subclavian vein – Systemic circulation
    4. Sentinel node – Right lumbar trunk – Cisterna chyli – Right bronchomediastinal trunk – Right lymphatic duct – Right subclavian vein – Systemic circulation
    5. Sentinel node – Left lumbar trunk – Cisterna chyli – Right bronchomediastinal trunk – Right lymphatic duct – Right subclavian vein – Systemic circulation
    1. Follicular hyperplasia
    2. Paracortical hyperplasia
    3. Sinus hyperplasia
    4. Diffuse hyperplasia
    5. Mixed B and T-cell hyperplasia
    1. Previous radiation therapy
    2. Female gender
    3. Axillary lymph node involvement
    4. Previous breast cancer
    5. Travel to Africa
    1. Recent immigration from equatorial Africa
    2. Patient gender
    3. Patient ethnicity
    4. Close family member with HIV
    5. Infected ulceration
    1. Kidney damage
    2. Hepatomegaly
    3. Splenomegaly
    4. Peripheral neuropathy
    5. Raynaud’s phenomenon
    1. Multiple myeloma
    2. Waldenstrom’s macroglobulinemia
    3. Chronic lymphocytic leukemia
    4. Chronic myelocytic leukemia
    5. Acute myelocytic leukemia
    1. Plasmapheresis
    2. Rituximab
    3. Cyclophosphamide
    4. Prednisone
    5. Vincristine
    1. Multiple Myeloma
    2. B-thalassemia
    3. Infectious mononucleosis
    4. Budd-Chiari Syndrome
    5. Myelofibrosis
    1. Pneumococcal septicemia
    2. Staphylococcal septicemia
    3. Thrombocytopenia
    4. Leukopenia
    5. Anemia
    1. Chemotherapy and autologous stem cell transplant
    2. Chemotherapy alone
    3. Renal dialysis
    4. Bisphosphonates
    5. Palliative care
    1. Multiple myeloma
    2. Metastatic bone disease
    3. POEMS syndrome
    4. Waldenstrom macroglobulinemia
    5. Monoclonal gammopathy of unknown significance
    1. Decreased ADAMTS13 causing platelet adhesion and formation of microthrombi
    2. Systemic activation of blood coagulation
    3. E. coli mediated endothelial damage and formation of microthrombi
    4. GPIIb/IIIa deficiency and failure of platelet aggregation
    5. Antiplatelet antibodies
    1. Plasma exchange
    2. Intravenous immunoglobulin
    3. Rehydration
    4. Platelet transfusion
    5. Renal dialysis
    1. >1.5 %
    2. <0.5 %
    3. 0.5 – 1.5 %
    4. 0 %
    1. β-thal major
    2. α-thal trait
    3. HbH disease
    4. Hb Bart disease
    5. β-thal minor
    1. B6
    2. B9
    3. B12
    4. B3
    5. B2
    1. 1.5 %
    2. 3.1 %
    3. 0.4 %
    4. 0.1 %
    5. 0.6 %
    1. Bone marrow hyperplasia
    2. Elevated lactose dehydrogenase
    3. Hemoglobinuria
    4. Decreased serum haptoglobin
    5. Hemosiderin
    1. Spectrin
    2. Decay accelerating factor
    3. Glucose-6-phosphatase dehydrogenase
    4. Iron absorption
    5. Glycosylphosphatidylinositol
    1. C55
    2. CD19
    3. CD3
    4. CD40L
    5. CD18
    1. An individual needs two defective β-globin genes to have the sickle cell trait
    2. Salmonella paratyphi can cause osteomyelitis in these patients
    3. Extravascular hemolysis
    4. Target cells are seen in blood smear
    5. Complications are due to vaso-occlusion
    1. I, IV
    2. I, II
    3. III, IV
    4. I, II, IV
    5. I, IV, V
    1. Red blood cells bind to IgG in warm temperatures >37 °C
    2. It is similarly associated with infectious mononucleosis
    3. Treatment consists of IVIG
    4. Red blood cells bind to IgM in cold temperatures >37 °C
    5. An indirect Coombs test will be positive
    1. A blood count will contain band forms, metamyelocytes, myelocytes.
    2. This condition can lead to chronic myelocytic leukemia.
    3. Myeloblasts, promyelocytes are expected to be found.
    4. The patient may develop anemia secondary to infection.
    5. Chemotherapy is the treatment of choice.
    1. III, IV, V
    2. I, III
    3. II, IV, V
    4. I, II, III
    5. I, II, IV
    1. I, II, V
    2. I, III, IV
    3. IV, V, VI
    4. I, II, III
    5. I, II, IV, V
    1. Graves Disease
    2. Cytomegalovirus infection
    3. Viral hepatitis
    4. Mononucleosis
    5. Toxoplasmosis
    1. The infectious organism can become latent in epithelial cells.
    2. The infectious organism can become latent in B cells.
    3. The infectious organism is Heterophile-positive.
    4. The infectious organism causes Burkitt lymphoma.
    5. Splenomegaly is a common finding.
    1. I, II, IV, V
    2. III, IV
    3. I, III, V
    4. I, II, III
    5. III, V
    1. The patient can be treated with a vitamin A derivative
    2. Auer Rods are responsible for gum hyperplasia and bleeding
    3. This condition is also developed early in life in patients with Down Syndrome
    4. Philadelphia chromosome may be seen and indicates a poor prognosis
    5. Myelodysplastic syndromes may give rise to the condition
    1. Hemolytic-uremic syndrome
    2. Salmonella infection
    3. Henoch-Schonlein purpura
    4. Systemic vasculitis
    5. Disseminated intravascular coagulation
    1. Heparin induced thrombocytopenia
    2. Immune thrombocytopenia
    3. DIC
    4. Myelodysplasia
    5. Thrombotic microangiopathy
    1. Lupus anticoagulant
    2. Antithrombin III
    3. Factor V Leiden
    4. Protein C
    5. Protein S
    1. Benign neutropenia
    2. Acute lymphoblastic leukemia
    3. Chronic lymphoblastic leukemia
    4. Aplastic anemia
    5. Sepsis
    1. Chronic kidney disease
    2. Liver disease
    3. Colorectal cancer
    4. Alcoholism
    5. Acute bleeding
    1. Chronic lymphocytic leukemia
    2. Acute lymphoblastic leukemia
    3. Immune thrombocytopenic purpura
    4. Aplastic anemia
    5. Tuberculosis of the bone marrowImmune thrombocytopenic purpura is a disease where thrombocytes- specific antibodies cause the destruction of thrombocytes. This would cause thrombocytopenia and this patient only has lymphocytosis in his CBC. Thus, this answer is not correct. Immune thrombocytopenic purpura is a disease where thrombocytes- specific antibodies cause the destruction of thrombocytes. This would cause thrombocytopenia and this patient only has lymphocytosis in his CBC. Thus, this answer is not correct.
    1. Bone marrow examination
    2. Repeated CBCs for several weeks and reassess
    3. Treatment for acute leukemia
    4. Treatment with corticosteroids
    5. Treatment with antibiotics

    Author of lecture Hematology Question Set 2

     Lecturio USMLE

    Lecturio USMLE


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