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Microcytic Anemia: Alpha- and Beta-Thalassemia – Red Blood Cell Pathology

by Carlo Raj, MD
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    About the Lecture

    The lecture Microcytic Anemia: Alpha- and Beta-Thalassemia – Red Blood Cell Pathology by Carlo Raj, MD is from the course Microcytic Anemia – Red Blood Cell Pathology (RBC). It contains the following chapters:

    • Alpha Thalassemia - Etiology
    • Alpha Thalassemia - Hemoglobin Electrophoresis
    • Alpha Thalassemia - Pathogenesis
    • Alpha Thalassemia - Clinical Pathology
    • Beta Thalassemia - Etiology and Pathogenesis
    • Beta Thalassemia - Morphology and Hemoglobin Electrophoresis
    • Beta Thalassemia - Clinical Pathology
    • Summary of Microcytic Anemias

    Included Quiz Questions

    1. Alpha and Gamma chains
    2. Alpha and Sigma chains
    3. Gamma and Sigma chains
    4. Beta and Gamma chains
    5. Alpha and Beta chains
    1. Alpha
    2. Beta
    3. None of these
    4. Sigma
    5. Gamma
    1. African Americans
    2. Caucasians
    3. Chinese
    4. Hispanics
    5. Mediterranean
    1. Hemoglobin H disease
    2. None of these
    3. Alpha thalassemia trait (Trans pattern)
    4. Alpha thalassemia trait (Cis pattern)
    5. Silent carrier
    1. 4 Beta globin chains
    2. 4 Alpha globin chains
    3. 1 Alpha and 3 Beta globin chains
    4. 2 Alpha and 2 Gamma globin chains
    5. 2 Alpha and 2 Beta globin chains
    1. 2 Alpha and 2 Delta globin chains
    2. 2 Gamma and 2 Delta globin chains
    3. 2 Beta and 2 Delta globin chains
    4. 2 Alpha and 2 Gamma globin chains
    5. 2 Alpha and 2 Beta globin chains
    1. Hemoglobin A
    2. Hemoglobin A2
    3. Hemoglobin Barts
    4. Hemoglobin H
    5. Hemoglobin F
    1. 4 Gamma globin chains
    2. 4 Beta globin chains
    3. 2 Beta and 2 Delta globin chains
    4. 2 Alpha and 2 Gamma globin chains
    5. 2 Alpha and 2 Beta globin chains
    1. Extramedullary hematopoiesis
    2. None of these
    3. Increased hemolysis
    4. Decreased production of erythrocytes
    5. Increased production of erythropoietin
    1. Hemoglobin H disease
    2. Alpha thalassemia trait (Trans pattern)
    3. Hemoglobin Barts disease
    4. Silent carrier of alpha thalassemia
    5. Alpha thalassemia trait (Cis pattern)
    1. Hemoglobin Barts
    2. Hemoglobin H
    3. Hemoglobin F
    4. Hemoglobin A2
    5. Hemoglobin S
    1. Hemoglobin A
    2. None of these
    3. Hemoglobin Barts
    4. Hemoglobin F
    5. Hemoglobin A2
    1. Chromosome 11
    2. Chromosome 21
    3. Chromosome 18
    4. Chromosome 13
    5. Chromosome 16
    1. Splice site mutation
    2. Silent mutation
    3. Missense mutation
    4. Nonsense mutation
    5. Frameshift mutation
    1. Cooley’s anemia
    2. Macrocytic anemia
    3. Silent carrier of alpha thalassemia
    4. Alpha thalassemia trait
    5. Beta thalassemia minor
    1. Hemoglobin A2
    2. Hemoglobin H
    3. Hemoglobin Barts
    4. Hemoglobin F
    5. Hemoglobin A
    1. It does not require beta chains for its synthesis.
    2. It is a subcategory of fetal hemoglobin F.
    3. It requires beta chain for its synthesis.
    4. It is a normal variant of hemoglobin A.
    5. It is the major type of hemoglobin synthesized overall.
    1. Hemoglobin F
    2. Hemoglobin A
    3. Hemoglobin A2
    4. Hemoglobin S
    5. Hemoglobin H
    1. Secondary hemochromatosis
    2. Esophageal web
    3. Hypertrophic obstructive cardiomyopathy
    4. Wilson disease
    5. Primary hemochromatosis
    1. Heart disease
    2. Vascular insufficiency
    3. Stroke
    4. Liver disease
    5. Renal failure
    1. Diastolic heart failure
    2. Bony changes
    3. Stunted growth
    4. High output cardiac failure
    5. Increased blood viscosity
    1. Ferritin
    2. RBC count
    3. None of these
    4. Serum Iron
    5. MCV

    Author of lecture Microcytic Anemia: Alpha- and Beta-Thalassemia – Red Blood Cell Pathology

     Carlo Raj, MD

    Carlo Raj, MD


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    very clear concepts explained
    By HARJOT C. on 13. January 2017 for Microcytic Anemia: Alpha- and Beta-Thalassemia – Red Blood Cell Pathology

    very well explained...best explanation very simple but clear concepts