Clinical Application of Renal Blood Flow (Part 2)

by Carlo Raj, MD

Questions about the lecture
My Notes
  • Required.
Save Cancel
    Learning Material 2
    • PDF
      Slides RenalBloodFlow2 RenalPathology.pdf
    • PDF
      Download Lecture Overview
    Report mistake

    About the Lecture

    The lecture Clinical Application of Renal Blood Flow (Part 2) by Carlo Raj, MD is from the course Renal Diagnostics. It contains the following chapters:

    • Changes in Glomerular Dynamics
    • Renin-Angiotensin-Aldosterone-System

    Included Quiz Questions

    1. Increase in hydrostatic pressure in bowmans space.
    2. Increase in hydrostatic pressure in the glomerular capillaries.
    3. Increase in renal plasma flow.
    4. Vasodilation of afferent arteriole.
    5. Increased glomerular filtration rate.
    1. All are contraindicated
    2. ACE inhibitors
    3. Angiotensin receptor blocker
    4. COX2 inhibitors
    5. Beta blockers
    1. Angiotensin II
    2. Renin
    3. Angiotensinogen
    4. Prostaglandin
    5. Aldosterone
    1. NSAIDs – vasoconstriction of afferent arteriole.
    2. Prostaglandins – vasoconstriction of the afferent arteriole.
    3. All are correct.
    4. COX 2 inhibitors – vasodilation of the efferent arteriole.
    5. Angiotensin II – vasodilation of efferent arteriole.
    1. It results in excess cortisol.
    2. It results in excess aldosterone.
    3. It is a tumor of the adrenal gland.
    4. It often presents with patients in alkalotic state.
    5. It often presents with secondary hypertension.
    1. All are correct.
    2. Renin released from juxtaglomerular cells.
    3. Angiotensin I is formed in the blood by renin stimulation.
    4. Angiontensin converting enzyme in the lung forms angiotensin II.
    5. Angiotensinogen formed in the liver.
    1. Metabolism of bradykinin.
    2. Stimulates ADH release from posterior pituitary.
    3. Release of aldosterone from the zona glomerulosa
    4. Stimulation of the hypothalamic thirst center.
    5. Na+/H+ activity in the proximal convoluted tubule.
    1. Addison’s disease,
    2. Reninoma.
    3. Cushing’s disease.
    4. Renal artery stenosis.
    5. Conn’s syndrome.
    1. Sarcoidosis
    2. Addison’s disease
    3. Angioedema.
    4. Renal artery stenosis.
    5. Tumor of the juxtaglomerular apparatus
    1. Decreases water reabsorption in the nephron.
    2. ENac upregulation on the apical membrane in the collecting duct.
    3. Increases sodium absorption overall.
    4. Stimulates the Na/K+ pump on the basolateral membrane stimulating K+ release into the lumen..
    5. Stimulates secretion of hydrogen ions in the collecting duct.

    Author of lecture Clinical Application of Renal Blood Flow (Part 2)

     Carlo Raj, MD

    Carlo Raj, MD

    Customer reviews

    5,0 of 5 stars
    5 Stars
    4 Stars
    3 Stars
    2 Stars
    1  Star