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Laboratoy Diagnostics: Arterial Blood Gas – Acidosis and Alkidosis (Step 3-5)

by Carlo Raj, MD
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    About the Lecture

    The lecture Laboratoy Diagnostics: Arterial Blood Gas – Acidosis and Alkidosis (Step 3-5) by Carlo Raj, MD is from the course Pulmonary Diagnostics. It contains the following chapters:

    • Step 3
    • Step 3: cont'd
    • Step 3: a
    • Step 4: AGMA
    • Step 5: Review Differential Dx.

    Included Quiz Questions

    1. Immediate respiratory compensation that will not correct pH
    2. Delayed respiratory compensation that will correct the pH
    3. Immediate metabolic compensation that will not correct pH
    4. Immediate respiratory compensation that will correct the pH
    5. Delay metabolic compensation that will correct the pH
    1. Bicarbonate binds to H+ ions and acts as a buffer
    2. Bicarbonate is excreted faster in acidic environments
    3. Metabolic acidosis leads to decreased production of bicarbonate
    4. Bicarbonate is lost, either through diarrhea or vomitting
    5. Acidic pH denatures bicarbonate
    1. Metabolic acidosis
    2. Respiratory acidosis
    3. Metabolic alkalosis
    4. Respiratory alkalosis
    5. PCO2 does not effect pH
    1. PCO2 = 1.5 x [HCO3-] + 8
    2. PCO2 = 15 x [HCO3-] + 8
    3. PCO2 = 0.9 x [HCO3-] + 9
    4. PCO2 = 0.9 x [HCO3-] + 16
    5. PCO2 = 0.9 x [HCO3-] + 8
    1. 60mmHg
    2. 10mmHg
    3. 40mmHg
    4. 80mmHg
    5. 30mmHg
    1. Respiratory acidosis with chronically increased bicarbonate
    2. Respiratory acidosis with acutely increased bicarbonate
    3. Respiratory alkalosis with chronically increased bicarbonate
    4. Metabolic alkalosis due to acute increased bicarbonate
    5. Respiratory acidosis due to chronically decreased bicarbonate
    1. Increased pH, decreased PCO2, decreased bicarbonate
    2. Decreased pH, decreased PCO2, decreased bicarbonate
    3. Increased pH, decreased PCO2, increased bicarbonate
    4. Increased pH, increased PCO2, decreased bicarbonate
    5. Increased pH, increased PCO2, increased bicarbonate
    1. 10-14
    2. 12-18
    3. 1-5
    4. 3-7
    5. 16-22
    1. 7-11
    2. 10-14
    3. 13-17
    4. 11-15
    5. 7-14
    1. Is not effected by levels of K or Mg in the body
    2. Anion gap = unmeasured anions - unmeasured cations
    3. Anion gap = Na - (Cl + HCO3)
    4. May include organic anions and proteins
    5. Is influenced by the levels of albumin in the serum
    1. 22
    2. 10
    3. 42
    4. 12
    5. 24
    1. Ketones
    2. Lactic acid
    3. Pyruvate
    4. Salicylic acid
    5. Hydrochloric acid
    1. Measured osmolality - calculated osmolality
    2. Urine osmolality - serum osmolality
    3. Calculated osmolality - measured osmolality
    4. Measured osmolality - (ketones + BUN)
    5. Na + glucose/2 + BUN
    1. Osmolal gap > 10
    2. Negative toxin screen
    3. Elevated ketones in urine
    4. Increased BUN
    5. Anion gap > 20
    1. Change in HCO3 = change in anion gap
    2. Change in HCO3 > change in anion gap
    3. Change in HCO3 < change in anion gap
    4. Change in delta gap = change in anion gap
    5. Change in delta gap > change in anion gap
    1. Bicarbonate being lower than expected in relation to the unmeasured anions calculated from the anion gap
    2. Normal anion gap with increased unmeasured anions above that expected due to change in bicarbonate
    3. Change in bicarbonate is lower than expected when compared to measured anions
    4. Change in bicarbonate equals change in anion gap
    5. Change in delta gap equals change in anion gap
    1. The acidosis is caused by loss of bicarbonate at the level of the kidney
    2. The acidosis is due to ingestion of an exogenous substance
    3. The acidosis is due to an increased production of bicarbonate at the level of the kidney
    4. The patient is compensating for the acidosis by secreting bicarbonate in the urine
    5. The acidosis has an extrarenal etiology
    1. Acetazolamide
    2. Spironolactone
    3. Addison's Disease
    4. Secondary Hypoaldosteronism
    5. RTA type IV
    1. Respiratory acidosis
    2. Respiratory alkalosis
    3. Metabolic alkalosis
    4. Anion gap metabolic acidosis
    5. Non-anion gap metabolic acidosis
    1. Diuretics
    2. Cushing's syndrome
    3. Exogenous alkali load
    4. Gitelman's syndrome
    5. Black licorice ingestion

    Author of lecture Laboratoy Diagnostics: Arterial Blood Gas – Acidosis and Alkidosis (Step 3-5)

     Carlo Raj, MD

    Carlo Raj, MD


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